21 Posts about #toxic_metabolic_encephalopathy Each one containers 🫙 Etiology Clinical features Radiological signs Treatment 1️⃣ Wernicke's encephalopathy https://m.facebook.com/story.php?story_fbid=832001682499736&id=100070697355985&mibextid=Nif5oz 2️⃣ Methanol poisoning https://m.facebook.com/story.php?story_fbid=832047512495153&id=100070697355985&mibextid=Nif5oz 3️⃣ Carbon monoxide poisoning https://www.facebook.com/share/p/16Mfd3tw1n/ 4️⃣ Vigabatrine associated toxicity https://www.facebook
21 Posts about #toxic_metabolic_encephalopath…
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USMLE High-Yield Questions on Instagram: "Bro think he unlocked flight mode 😭✈️ ⸻ Explanation 🧠🔥 This is acute psychosis with hallucinations, delusions, and severe insomnia without intoxication. The first-line treatment for acute agitation with psychosis is an antipsychotic, especially a high-potency typical like haloperidol. Haloperidol rapidly controls: • Hallucinations • Delusions • Severe agitation • Dangerous behavior Vitals are stable → no benzodiazepine is needed unless withdrawal is s
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INTRANUCLEAR OPHTHALMOPLEGIA During horizontal gaze, the MLF on each side of the brain stem enables abduction of one eye to be coordinated with adduction of the other. The MLF connects the following structures: Sixth cranial nerve nucleus (which controls the lateral rectus, responsible for abduction) Adjacent horizontal gaze center (paramedian pontine reticular formation) Contralateral third cranial nerve nucleus (which controls the medial rectus, responsible for adduction) The MLF also connects
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